Max doesn't do repeats, and he definitely doesn't do commitments. Months after a hot encounter with a smooth-talking stranger in a bar room bathroom, Max comes face to face with the same sexy doctor during the unveiling of the community center's dance studio. And the man is just as pushy and perceptive as Max remembers. Because of their mutual friendships, Max now has to work overtime to keep Dr. Ashton Jackson at arm's length. Will he succeed in resisting the undeniable attraction he feels toward the man, or will he risk putting his heart on the line for love... again?Ash isn't afraid of love. He's just never met The One. But there was something about the brown-eyed stranger he hooked up with that he can't get out of his head. When their paths cross again unexpectedly, Ashton hopes to convince the wounded man with the heart of gold to give love a second chance. But what happens when the truth behind Max's heartache threatens to leave Ash questioning everything he's ever believed about himself? Will he too be left broken? Or will he and Max survive the break?**An M/M romance and book two in the Chadwick trilogy. Can be read as a standalone and contains explicit sex and graphic language.

My name is Justin, and when I was seventeen, I fell in love with a boy named Damon.Damon was sick, only I didn't know it at the time. And like any disease left untreated, it festered and burrowed into the unaffected areas of us, until we were both so far from healthy, we were killing one another. Then a tragedy sparked our implosion.With nothing of us remaining to salvage, I was left to pick up the pieces, while Damon found his escape in the abyss. Leaving me alone with Blake.Damon and Blake are two halves of a whole. But Damon is the coldest part of darkness, and Blake the warmest part of light. Which form of ecstasy would you choose?I love Blake with a ferocious intensity akin to the peeling back of skin. But Blake isn't Damon. And Damon was gone--until now.What once threatened to tear us apart has become our catharsis.Outnumbered in the relationship with the man who was my life, I've begun to learn more about love than I ever thought possible.An M/M romance and book one in the Chadwick trilogy.Can be read as a standalone, and does not end on a cliffhanger. Surviving the Merge contains explicit sex and graphic language, deals with mental illness, does not contain cheating, and ends with a HEA.Author's note: For a full set of trigger warnings, please use the Amazon "look inside" feature to find them within the first several pages.

This book provides a qualitative and quantitative exploration of the action of radiation on living matter which leads to a complete and coherent interpretation of radiation biology. It takes readers from radiation-induced molecular damage in the nucleus of the cell and links this damage to cellular effects such as cell killing, chromosome aberrations and mutations before exploring organ damage, organism lethality and cancer induction. It also deals with radiological protection concepts and the difficulties of predicting the dose–effect relationship for low-dose and dose rate radiation risk. The book ends with separate chapters dealing with the effects of UV light exposure and risk classification of chemical mutagens, both of which are derived by logical extensions of the radiation model. This book will provide the basic foundations of radiation biology for undergraduate and graduate students in medical physics, biomedical engineering, radiological protection, medicine, radiology and radiography. Features Presents a comprehensive insight into radiation action on living matter Contains important implications for radiological protection and regulations Provides analytical methods for applications in radiotherapy

Imagine an eighteen-year-old American girl who has never read a newspaper, watched television, or made a phone call. An eighteen-year-old-girl who has never danced—and this in the 1960s. It is in Cambridge, Massachusetts where Leonard Feeney, a controversial (soon to be excommunicated) Catholic priest, has founded a religious community called the Slaves of the Immaculate Heart of Mary. The Center's members—many of them educated at Harvard and Radcliffe—surrender all earthly possessions and aspects of their life, including their children, to him. Patricia Chadwick was one of those children, and Little Sister is her account of growing up in the Feeney sect. Separated from her parents and forbidden to speak to them, Patricia bristles against the community’s draconian rules, yearning for another life. When, at seventeen, she is banished from the Center, her home, she faces the world alone, without skills, family, or money but empowered with faith and a fierce determination to succeed on her own, which she does, rising eventually to the upper echelons of the world of finance and investing. A tale of resilience and grace, Little Sister chronicles, in riveting prose, a surreal childhood and does so without rancor or self-pity.

Glutton, demon of destruction, symbol of slaughter, mightiest of wilderness villains… The wolverine comes marked with a reputation based on myth and fancy. Yet this enigmatic animal is more complex than the legends that surround it. With a shrinking wilderness and global warming, the future of the wolverine is uncertain. The Wolverine Way reveals the natural history of this species and the forces that threaten its future, engagingly told by Douglas Chadwick, who volunteered with the Glacier Wolverine Project. This five-year study in Glacier National Park – which involved dealing with blizzards, grizzlies, sheer mountain walls, and other daily challenges to survival – uncovered key missing information about the wolverine’s habitat, social structure and reproduction habits. Wolverines, according to Chadwick, are the land equivalent of polar bears in regard to the impacts of global warming. The plight of wolverines adds to the call for wildlife corridors that connect existing habitat that is proposed by the Freedom to Roam coalition.

In late 1971 we were involved in a study of the interaction of radiation with matter and were trying to use measurements of radiation fluorescence in biological molecules to indicate how radiation affected living cells. It soon became apparent that we were working in the dark; the doses we used to get a significant signal were too large to be of interest for radiation biology and although the DNA molecule appeared to be the most likely target molecule we did not know which sort of events and which sort oflesions were the most important. We decided to alter our approach to see if we could find any consistent mathematical order in the radiobiological dose relationships. We found that cell survival curves could be very usefully described by a linear-quadratic dose relationship and very soon came to the somewhat premature but, as it turned out, most effective conclusion that the induction of DNA double strand breaks should be linear-quadratic. In deciding that the DNA double strand break was the crucial and all-important lesion we were able to associate the mathematical analysis with the biology of the cell and were able to relate known properties of the DNA molecule to known radiobiological effects. On the other hand, we were restricted and brought, from an abstract two-hit lesion which could have any property one wished, down to earth, to a defined moleccular structure of nanometer dimensions and well-known functions and properties.

Advances in Radiation Biology, Volume 7, provides an overview of the state of knowledge in the field of radiation biology. The book contains six chapters and opens with a study on the exploitation of the unique properties of purified lesion-recognizing enzymes as the basis of assays that monitor the repair of DNA lesions in vivo. This is followed by separate chapters on the effects of radiation on living eukaryotic cells; the ecological effects of ionizing radiation; and the applicability of bacterial models of DNA repair and recovery to UV-irradiated mammalian cells. Subsequent chapters focus on acute radiation effects on the adult nervous system and the repair of DNA modified by cytotoxic, mutagenic, and carcinogenic chemicals.